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Date: Tue, 27 Feb 2018 12:23:10 -0500
From: "Barbara  Mendez" <barbara..mendez@dwuilntb.com>
To:   <lojban@lojban.org>
Subject: You shouldn't have to live with-pain: #2136904983
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 Content preview:  ============================================ Health-Newsletter
    No. 27781 ============================================ 27 - FEB - 2018 ============================================
    No one should have to [...] 
 
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============================================
Health-Newsletter No. 27781
============================================
27 - FEB - 2018
============================================
No one should have to live in-pain
============================================
Greetings lojban@lojban.org,

Do you have a burning-feeling or pins-and-needles constantly?

This could be a sign of-neuropathy, which is typically-brought about by-diabetes. 

In the past, it was believed there was "nothing" anyone could do about it. . . but there is now!

You shouldn't have to live with-pain, and healthy-nerves shouldn't feel like constant-burning. 


Find-out more here: http://www.dwuilntb.com/McDowell-simulators/5d08j6Fzs236C84MxhvLKgFxhvLKgzhkr3a5


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This definition conveys the important message that not all patients with peripheral nerve dysfunction have a neuropathy caused by diabetes. Confirmation can be established with quantitative electrophysiology, sensory, and autonomic function testing. 1) Acute sensory neuropathy. Acute sensory neuropathy is rare, tends to follow periods of poor metabolic control (e.g., ketoacidosis) or sudden change in glycemic control (e.g., &ldquo;insulin neuritis&rdquo;), and is characterized by the acute onset of severe sensory symptoms (as detailed below) with marked nocturnal exacerbation but few neurologic signs on examination of the legs. 2) Chronic sensorimotor DPN. This is the most common presentation of neuropathy in diabetes, and up to 50% of patients may experience symptoms, most frequently burning pain, electrical or stabbing sensations, parasthesiae, hyperasthesiae, and deep aching pain.

asymmetric motor deficits, progressive sensory neuropathy in spite of optimal glycemic control together with typical electrophysiological findings, and an unusually high cerebro-spinal fluid protein level all suggest the possibility of an underlying treatable demyelinating neuropathy. As immunomodulatory therapy with combinations of corticosteroids, plasmapheresis, and intravenous immune globulin can produce a relatively rapid and substantial improvement in neurological deficits and electrophysiology in some cases of CIDP, referral to a neurologist is indicated if this diagnosis is suspected. Diabetic autonomic neuropathy (DAN) results in significant morbidity and may lead to mortality in some patients with diabetes. The most common dysautonomic features are listed in Table 2, together with their associated symptoms and management. The symptoms of autonomic dysfunction should be elicited carefully during the history, particularly since many of these symptoms are potentially treatable.

Prevalence data for DAN range from 1.6 to 90% depending on tests used, populations examined, and type and stage of disease. Risk factors for the development of DAN include diabetes duration, age, and long-term poor glycemic control. DAN may cosegregate with factors predisposing to macrovascular events such as raised blood pressure and dyslipidemia. Thus, in addition to good glycemic control, lipid modulation and blood pressure control may be beneficial in the prevention of DAN. There are no true population-based studies using radioisotopic techniques that quantify gastric emptying in diabetic patients, but cross-sectional studies have indicated that ?50% of outpatients with long-standing diabetes have delayed gastric emptying and up to 76% of diabetic outpatients indicate that they have one or more gastrointestinal symptom, the most common of which is constipation. Both upper- and lower-gastrointestinal symptoms occur more frequently in individuals with diabetes than in control subjects, but the symptoms are nonspecific and occur in the general population. Genitourinary bladder dysfunction has been shown in 43&ndash;87% of individuals with type 1 diabetes.


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